Anti-Smad3 antibody (Ab28379)
MADH3, SMAD3, SMAD family member 3, SMAD 3, hSMAD3, JV15-2, Mothers against decapentaplegic homolog 3, MAD homolog 3, Mad3, Mothers against DPP homolog 3, hMAD-3
反应种属：Human
抗原名称：SMAD3
应用：IHC
详细信息
免疫原The exact immunogen used to generate this antibody is proprietary information.
宿主/亚型Rabbit / IgG
抗体类型Polyclonal
产品形式Liquid
纯化方式Affinity purification Immunogen
储存缓冲液pH: 7.6 Preservative: 0.1% Sodium azide Constituents: PBS, 1% BSA
储存条件运输条件: Blue Ice
推荐的短期储存时间: 1-2 weeks
推荐的短期储存条件: +4°C
推荐的长期储存条件: -20°C
储存信息: Avoid freeze / thaw cycle
无载体蛋白No
文献引用 (152)
Palmitoylation Induced Activation of SMAD3 Exacerbates Colitis by Promoting Tissue-resident Memory T Cells Differentiation.
棕榈酰化诱导的 SMAD3 活化通过促进组织驻留记忆 T 细胞分化加剧结肠炎。
Runx2 Regulated Airway Homeostasis Is Disrupted in Asthma.
Runx2 调节的气道稳态在哮喘中受到破坏。
Myofibroblast lineage mapping and inhibiting subretinal fibrosis by targeting SMAD3 and MRTF pathways via microRNA-24 functional study.
通过microRNA-24功能研究，靶向SMAD3和MRTF通路，绘制肌成纤维细胞谱系图并抑制视网膜下纤维化。
Gain-of-function variants in SMAD4 compromise respiratory epithelial function.
SMAD4 的功能获得性变异会损害呼吸道上皮功能
SMAD2 S-palmitoylation promotes its linker region phosphorylation and T(H)17 cell differentiation in a mouse model of multiple sclerosis.
在多发性硬化症小鼠模型中，SMAD2 S-棕榈酰化促进其连接区磷酸化和 T(H)17 细胞分化
Therapeutic potential of formononetin in cirrhotic portal hypertension: modulating hepatic fibrosis, macrophage polarization, and lymphangiogenesis.
芒柄花素在肝硬化门静脉高压症中的治疗潜力：调节肝纤维化、巨噬细胞极化和淋巴管生成
Furin Drives Colorectal Cancer Progression and Chemoresistance Through the TGF-β/ERK Signaling Pathway.
Furin 通过 TGF-β/ERK 信号通路驱动结直肠癌进展和化疗耐药性。
Adenosine A2B Receptor Antagonism Interferes with TGF-β Cellular Signaling Through SMAD2/-3 and p65-Nf-κB in Podocytes and Protects from Phenotypical Transformation in Experimental Diabetic Glomerulopathy
腺苷A2B受体拮抗剂通过SMAD2/-3和p65-NF-κB干扰足细胞中的TGF-β细胞信号传导，并保护实验性糖尿病肾小球病免受表型转化。
USP15-dependent modulation of TGF-β/Smad2/3 signaling: implications for chondrocyte matrix degradation and autophagy in osteoarthritis.
USP15 依赖性 TGF-β/Smad2/3 信号调节：对骨关节炎中软骨细胞基质降解和自噬的影响。
TGF-β2 enhances nanoscale cortex stiffness via condensation of cytoskeleton-focal adhesion plaque.
TGF-β2 通过细胞骨架-粘着斑的凝聚增强纳米级皮质刚度
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