Surface protein glycosylation conserved in the human pathogen Mycoplasma genitalium and retained in the synthetic organism JCVI-Syn3A

人类病原体生殖支原体中保守的表面蛋白糖基化,在合成生物体JCVI-Syn3A中也得以保留。

阅读:1

Abstract

Protein glycosylation has been reported in all forms of life. The genus Mycoplasma is composed of highly genome-streamlined bacterial symbionts, making them model organisms for investigating minimal genome concepts. Previous work from our group showed mycoplasmas scavenge hexoses from exogenous oligosaccharides to glycosylate surface proteins at serine, threonine, asparagine, and glutamine residues without utilizing a consensus sequence as seen in canonical glycosylation systems. We report here that this surface protein hexosylation system is conserved in Mycoplasma genitalium, a human urogenital pathogen with a 580-kbp genome that can be cultured axenically. We also report this modification is found in the ruminant pathogen Mycoplasma mycoides subsp. capri and is conserved in JCVI-Syn3A, a nonpathogenic mycoplasma with a synthetic minimal M. mycoides genome containing genes that are essential for survival and robust growth under axenic culture conditions. In contrast to known glycoproteins, we have detected evidence of glycosylation of aspartic acid and glutamic acid residues, which expands the pool of potential glycosyl acceptors in bacteria to include the acidic amino acids.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。